We believe that many of the current approaches address only the nutritional and feed production technology, but for animals,there is no way to address the mechanical damage to the gut caused by artificial breeding through nutrition or changes to the feed production technology. ​These adjustments and changes deal only with the digestibility of the feed itself, but not with the integrity of the gut.

In Rota, the regulation of intestinal health through nutrition, immunity and microecology is approaching maturity. More valuable is, we provide directly solutions to the problems of intestinal epithelial damage and growth and developmental inhibition.
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Sow Protection

Health problems such as low feed intake, heat stress, low resistance, overweight or lean sows during pregnancy, inflammation or constipation, parity and improper prenatal and postnatal management can affect litter size and milk volume and ultimately the efficiency of farming.

Optimizing sow performance is fundamental to improving the health, welfare and productivity of sows and  progeny, leading to sustainable pig production.

For sows, the factors that affect reproductive performance are mainly manifested in three ways:

✦The developmental delay of ovarian follicles, the low maturity of oocytes and the low fertilization rate are mainly related to the quality of oocyte development.

✦Embryo implantation and early embryo development.

✦The secretion of progesterone in the corpus luteum of the ovary and the stability of pregnancy hormones.

Addition of endogenous bioactive factors can help regulate the trophoblastic function of follicular granulosa cells, promote oocyte development, and accommodate endometriosis regulation.

It promotes the production of progesterone in the corpus luteum of the ovary and maintains the stability of pregnancy. Learn more

Piglets Weanning Care
Early weaning of suckling piglets is usually carried out before 25 days of age, at the early stage of weaning, the intestinal enterocyte were severely damaged and the villus height was greatly reduced. The shortened villi are closely adhered to each other to form a smooth surface of the intestinal cavity, and the short, compact and smooth intestinal villi have a great reduction in the absorption efficiency of the nutrient substances, and the incomplete intestinal epithelial structure causes the risk of the infection of the disease to increase. The repair of intestinal epithelium after weaning should continue at least 4 Weeks after Weaning

Our Solution:

The addition of repair factor to the diet after weaning can make up for the lack of milk-derived growth factor, accelerate the repair of intestinal epithelial structure, promote the secretion of intestinal epithelial digestive enzymes, and play a direct role in the improvement of dyspepsia and the reduction of pathogenic infection.
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Diarrhea Control

The immature intestine of piglets has a large permeability, and the intestinal epithelial cells, which form the mechanical barrier, are relatively loose. Therefore, weaning stress, sudden changes in food structure, and common infections not only cause damage to the intestinal mucosa of piglets but also weaken the mechanical barrier function of the intestine.

Due to immature intestinal development, increased permeability and impaired barrier function, can ultimately lead to slow growth in the body caused by overstimulation by sensitive antigens.

Increased intestinal permeability is an important cause of microecology problems such as diarrhea in piglet intestines. The main manifestations are:

✦Increased risk of microbial invasion, and the contents of the intestine are more likely to spread to the submucosa, leading to inflammatory reactions.
✦The balance of digestive environment such as osmotic pressure and electrolyte between the intestinal lumen and basement will be destroyed, and the digestion and absorption of nutrients will be disrupted.
✦The concentration of fermented substrates in the hindgut may increase, causing an imbalance of the intestinal microecology.

Our solution aims to:

✦Promote the secretion of signaling molecules that mediate the repair and regeneration of damaged mucosa.

✦Regulate the structure of extracellular calcium-binding proteins to promote tight cell connections, reduce paracellular permeability, and improve intestinal barrier function.

✦Activate cell migration abilities, maintain stable environments such as intestinal osmotic pressure balance, and restore intestinal mucosal barrier function. Learn More